If you are not certain whether a specific product can be given to a child, or if you have any questions about the amount to give, check with your health care professional, especially if it contains: Antihistamines—Nightmares, unusual excitement, nervousness, restlessness, or irritability may be more likely to occur in children taking antihistamines. Also, unusual excitement or restlessness may be more likely to occur in children receiving these medicines. This is very important because salicylates may cause a serious illness called Reye's syndrome in children with fever caused by a virus infection, especially flu or chickenpox.
Also, children may be more sensitive to the aspirin or other salicylates contained in some of these medicines, especially if they have a fever or have lost large amounts of body fluid because of vomiting, diarrhea , or sweating. Geriatric TOP The elderly are usually more sensitive to the effects of this medicine, especially if it contains: Antihistamines—Confusion, difficult or painful urination , dizziness, drowsiness, feeling faint, or dryness of mouth, nose, or throat may be more likely to occur in elderly patients.
Also, nightmares or unusual excitement, nervousness, restlessness, or irritability may be more likely to occur in the elderly taking antihistamines. Also, increases in blood pressure may be more likely to occur in elderly persons taking decongestants.
Acetaminophen —Studies on birth defects have not been done in humans. However, acetaminophen has not been shown to cause birth defects or other problems in humans. Alcohol—Some of these combination medicines contain a large amount of alcohol. Too much use of alcohol during pregnancy may cause birth defects. Antihistamines—Antihistamines have not been shown to cause problems in humans. Caffeine—Studies in humans have not shown that caffeine causes birth defects.
However, studies in animals have shown that caffeine causes birth defects when given in very large doses amounts equal to the amount of caffeine contained in 12 to 24 cups of coffee a day.
Codeine —Although studies on birth defects with codeine have not been done in humans, it has not been reported to cause birth defects in humans. Though it helps many people, this product has a risk for abuse and may sometimes cause addiction. Take this product exactly as prescribed to lower the risk of addiction. Ask your doctor or pharmacist for more details. When used for an extended time, this medication may not work as well and may require different dosing.
Talk with your doctor if this medication stops working well. Tell your doctor if your condition persists for more than 1 week, if it worsens, or if it occurs with fever, rash , or persistent headache. Call your doctor for instructions. Symptoms of an overdose include: Extreme dizziness or weakness, shortness of breath, slow heartbeat, seizures, and cold, clammy skin. This medicine may make you dizzy or drowsy.
Avoid driving, using machines, or doing anything else that could be dangerous if you are not alert. Make sure any doctor or dentist who treats you knows that you are using this medicine.
Additionally, concurrent use of codeine with barbiturates may decrease codeine plasma concentrations, decrease opioid efficacy, and potentially lead to a withdrawal syndrome in those with physical dependence to opioid agonists. Monitor for signs of opioid withdrawal. Discontinuation of barbiturates may increase the risk of increased opioid-related adverse reactions, such as fatal respiratory depression. Barbiturates are inducers of CYP3A4, an isoenzyme partially responsible for the metabolism of codeine.
Acetaminophen; Butalbital; Caffeine; Codeine: Major Concomitant use of hydrocodone with other CNS depressants may lead to hypotension, profound sedation, coma, respiratory depression and death. Prior to concurrent use of hydrocodone in patients taking a CNS depressant, assess the level of tolerance to CNS depression that has developed, the duration of use, and the patient's overall response to treatment. Consider the patient's use of alcohol or illicit drugs.
Also consider a using a lower dose of the CNS depressant. Monitor patients for sedation and respiratory depression. Hypotension, profound sedation, coma, respiratory depression, or death may occur.
Prior to concurrent use of oxycodone in patients taking a CNS depressant, assess the level of tolerance to CNS depression that has developed, the duration of use, and the patient's overall response to treatment. Monitor for sedation and respiratory depression.
Major Avoid the concomitant use of pentazocine and opiate agonists, such as codeine. Pentazocine may cause withdrawal symptoms in patients receiving chronic opiate agonists. Concurrent use of pentazocine with other opiate agonists can cause additive CNS, respiratory, and hypotensive effects. The additive or antagonistic effects are dependent upon the dose of the opiate agonist used; antagonistic effects are more common at low to moderate doses of the opiate agonist. Major Concomitant use of tramadol increases the seizure risk in patients taking opiate agonists.
Also, tramadol can cause additive CNS depression and respiratory depression when used with opiate agonists; avoid concurrent use whenever possible. If used together, extreme caution is needed, and a reduced tramadol dose is recommended.
Therefore, psychotropic pharmacodynamic interactions could occur following concomitant administration of drugs with significant CNS or psychotropic activity such as opiate agonists. In addition, aldesleukin, IL-2, is a CYP3A4 inhibitor and may increase oxycodone plasma concentrations and related toxicities including potentially fatal respiratory depression.
If therapy with both agents is necessary, monitor patients for an extended period and adjust oxycodone dosage as necessary. Moderate Monitor for an increase in codeine-related adverse reactions including sedation and respiratory depression if coadministration with amlodipine is necessary; adjust the dose of codeine if necessary.
Amlodipine is a weak CYP3A4 inhibitor. Concomitant use may result in an increase in codeine plasma concentrations, resulting in greater metabolism by CYP2D6 and increased morphine concentrations. Moderate Opiate agonists may potentiate orthostatic hypotension when used concurrently with thiazide diuretics. Major Because of the potential risk and severity of serotonin syndrome, caution and careful monitoring are recommended when administering serotonin-receptor agonists with other drugs that have serotonergic properties such as opioids.
Serotonin syndrome is characterized by the rapid development of hyperthermia, hypertension, myoclonus, rigidity, autonomic instability, mental status changes e. Codeine and the serotonin-receptor agonist should be discontinued if serotonin syndrome occurs and supportive symptomatic treatment should be initiated. Major Patients taking medications that decrease GI motility may be at greater risk for serious complications from alosetron, like constipation, via a pharmacodynamic interaction.
Constipation is the most frequently reported adverse effect with alosetron. Major Concomitant use of opiate agonists with benzodiazepines may cause respiratory depression, hypotension, profound sedation, and death. Limit the use of opiate pain medications with benzodiazepines to only patients for whom alternative treatment options are inadequate. If an opiate agonist is initiated in a patient taking a benzodiazepine, use a lower initial dose of the opiate and titrate to clinical response.
If a benzodiazepine is prescribed for an indication other than epilepsy in a patient taking an opiate agonist, use a lower initial dose of the benzodiazepine and titrate to clinical response. Avoid prescribing opiate cough medications in patients taking benzodiazepines.
Moderate Patients should not take alvimopan if they have received therapeutic doses of opiate agonists for more than seven consecutive days immediately before initiation of alvimopan therapy.
Patients recently exposed to opioids are expected to be more sensitive to the effects of mu-opioid receptor antagonists and may experience adverse effects localized to the gastrointestinal tract such as abdominal pain, nausea, vomiting, and diarrhea.
Moderate The use of these drugs together must be approached with caution. Although commonly used together for additive analgesic effects, the patient must be monitored for respiratory depression, hypotension, and excessive sedation due to additive effects on the CNS and blood pressure.
In rare instances, serious morbidity and mortality has occurred. Limit the use of opiate pain medications with local anesthetics to only patients for whom alternative treatment options are inadequate. The use of the local anesthetic may allow for the use a lower initial dose of the opiate and then the doses can be titrated to proper clinical response.
Moderate Concomitant use of codeine with amiodarone may alter codeine plasma concentrations, resulting in an unpredictable effect such as reduced efficacy or symptoms of opioid withdrawal or prolonged opioid adverse reactions, including hypotension, respiratory depression, profound sedation, coma, and death. If coadministration is necessary, monitor patients closely at frequent intervals and consider a dosage adjustment of codeine until stable drug effects are achieved.
Discontinuation of amiodarone could alter codeine plasma concentrations, resulting in an unpredictable effect such as prolonged opioid adverse reactions or decreased opioid efficacy, and potentially lead to a withdrawal syndrome in those with physical dependence to codeine. If amiodarone is discontinued, monitor the patient carefully and consider adjusting the opioid dosage if appropriate.
Concomitant use may potentially lead to increased CNS depression, sedation, respiratory depression, or hypotensive responses. Both TCAs and opiate agonists may produce constipation. Use codeine with caution and in reduced dosages in patients taking TCAs. Moderate Concomitant use of central nervous system CNS depressants can potentiate the effects of codeine, which may potentially lead to respiratory depression, CNS depression, sedation, or hypotensive responses.
Examples of drugs associated with CNS depression include amoxapine. If concurrent use of codeine and another CNS depressant is imperative, reduce the dose of one or both drugs. Moderate Concomitant use of codeine with clarithromycin may increase codeine plasma concentrations, resulting in greater metabolism by CYP2D6, increased morphine concentrations, and prolonged opioid adverse reactions, including hypotension, respiratory depression, profound sedation, coma, and death.
If coadministration is necessary, monitor patients closely at frequent intervals and consider a dosage reduction of codeine until stable drug effects are achieved. Discontinuation of clarithromycin could decrease codeine plasma concentrations, decrease opioid efficacy, and potentially lead to a withdrawal syndrome in those with physical dependence to codeine.
If clarithromycin is discontinued, monitor the patient carefully and consider increasing the opioid dosage if appropriate.
Clarithromycin is a strong inhibitor of CYP3A4. Moderate Monitor patients for signs of urinary retention or reduced gastric motility when codeine is used concomitantly with an anticholinergic drug. Opiates increase the tone and decrease the propulsive contractions of the smooth muscle of the gastrointestinal tract. Prolongation of the gastrointestinal transit time may be the mechanism of the constipating effect. Moderate Concomitant use of codeine with apalutamide can decrease codeine levels, resulting in less metabolism by CYP2D6 and decreased morphine concentrations; this may result in decreased efficacy or onset of a withdrawal syndrome in patients who have developed physical dependence.
If coadministration is necessary, monitor for reduced efficacy of codeine and signs of opioid withdrawal; consider increasing the dose of codeine as needed. If apalutamide is discontinued, consider a dose reduction of codeine and frequently monitor for signs or respiratory depression and sedation. Apalutamide is a strong CYP3A4 inducer. Moderate Apomorphine causes significant somnolence. Concomitant administration of apomorphine and CNS depressants could result in additive depressant effects.
Minor Theoretically, apraclonidine might potentiate the effects of CNS depressant drugs such as opiate agonists. Although no specific drug interactions were identified with systemic agents and apraclonidine during clinical trials, apraclonidine can cause dizziness and somnolence.
Moderate Concomitant use of codeine with oral, multi-day regimens of aprepitant, fosaprepitant may increase codeine plasma concentrations, resulting in greater metabolism by CYP2D6, increased morphine concentrations, and prolonged opioid adverse reactions, including hypotension, respiratory depression, profound sedation, coma, and death. Discontinuation of aprepitant, fosaprepitant could decrease codeine plasma concentrations, decrease opioid efficacy, and potentially lead to a withdrawal syndrome in those with physical dependence to codeine.
If aprepitant, fosaprepitant is discontinued, monitor the patient carefully and consider increasing the opioid dosage if appropriate. Aprepitant, fosaprepitant, when administered as an oral, 3-day regimen, is a moderate inhibitor of CYP3A4. Moderate Concomitant use of codeine with lumefantrine may increase codeine plasma concentrations, but decrease the plasma concentration of the active metabolite, morphine, resulting in reduced efficacy or symptoms of opioid withdrawal. Discontinuation of lumefantrine could decrease codeine plasma concentrations and increase morphine plasma concentrations resulting in prolonged opioid adverse reactions, including hypotension, respiratory depression, profound sedation, coma, and death.
If lumefantrine is discontinued, monitor the patient carefully and consider reducing the opioid dosage if appropriate. Lumefantrine is a moderate inhibitor of CYP2D6. Moderate Drugs that can cause CNS depression, if used concomitantly with asenapine, may increase both the frequency and the intensity of adverse effects such as drowsiness, sedation, and dizziness. Caution should be used when asenapine is given in combination with other centrally-acting medications including opiate agonists.
Major Concomitant use of opiate agonists with skeletal muscle relaxants may cause respiratory depression, hypotension, profound sedation, and death. Limit the use of opiate pain medications with skeletal muscle relaxants to only patients for whom alternative treatment options are inadequate. If an opiate agonist is initiated in a patient taking a skeletal muscle relaxant, use a lower initial dose of the opiate and titrate to clinical response.
If a skeletal muscle relaxant is prescribed for a patient taking an opiate agonist, use a lower initial dose of the skeletal muscle relaxant and titrate to clinical response. Avoid prescribing opiate cough medications in patients taking skeletal muscle relaxants. Moderate Concomitant use of codeine with atazanavir may increase codeine plasma concentrations, resulting in greater metabolism by CYP2D6, increased morphine concentrations, and prolonged opioid adverse reactions, including hypotension, respiratory depression, profound sedation, coma, and death.
Discontinuation of atazanavir could decrease codeine plasma concentrations, decrease opioid efficacy, and potentially lead to a withdrawal syndrome in those with physical dependence to codeine.
If atazanavir is discontinued, monitor the patient carefully and consider increasing the opioid dosage if appropriate. Atazanavir is a strong inhibitor of CYP3A4. Moderate Concomitant use of codeine with cobicistat may increase codeine plasma concentrations, resulting in greater metabolism by CYP2D6, increased morphine concentrations, and prolonged opioid adverse reactions, including hypotension, respiratory depression, profound sedation, coma, and death.
Discontinuation of cobicistat could decrease codeine plasma concentrations, decrease opioid efficacy, and potentially lead to a withdrawal syndrome in those with physical dependence to codeine. If cobicistat is discontinued, monitor the patient carefully and consider increasing the opioid dosage if appropriate. Cobicistat is a strong inhibitor of CYP3A4. Moderate Concomitant use of codeine with other CNS depressants, such as neuromuscular blockers, can potentiate the effects of alfentanil on respiration, alertness, and blood pressure.
A dose reduction of one or both drugs may be warranted. Severe Codeine use is contraindicated in patients who are receiving or who have received monoamine oxidase inhibitors MAOIs within the previous 14 days. Methylene blue is a reversible inhibitor of MAO.
Concomitant use of codeine with other serotonergic drugs such as MAOIs may result in serious adverse effects including serotonin syndrome. MAOIs may cause additive CNS depression, respiratory depression, drowsiness, dizziness, or hypotension when used with opiate agonists such as codeine. Use caution during coadministration.
Reduced GI motility when combined with opiate agonists may increase the risk of serious GI related adverse events. Atropine; Hyoscyamine; Phenobarbital; Scopolamine: Belladonna Alkaloids; Ergotamine; Phenobarbital: Moderate The vagal effects and respiratory depression induced by opiate agonists may be increased by the use of benzonatate. Moderate Bethanechol facilitates intestinal and bladder function via parasympathomimetic actions.
Opiate agonists impair the peristaltic activity of the intestine. Thus, these drugs can antagonize the beneficial actions of bethanechol on GI motility. Moderate Concomitant use of codeine with bexarotene can decrease codeine levels, resulting in less metabolism by CYP2D6 and decreased morphine concentrations; this may result in decreased efficacy or onset of a withdrawal syndrome in patients who have developed physical dependence.
If bexarotene is discontinued, consider a dose reduction of codeine and frequently monitor for signs or respiratory depression and sedation. Bexarotene is a moderate CYP3A4 inducer. Bismuth Subcitrate Potassium; Metronidazole; Tetracycline: Moderate Additive constipation may be seen with concurrent use of opiate agonists and antidiarrheals.
Opioids increase the tone and decrease the propulsive contractions of the smooth muscle of the gastrointestinal tract. Bismuth Subsalicylate; Metronidazole; Tetracycline: Moderate Concomitant use of codeine with bosentan can decrease codeine levels, resulting in less metabolism by CYP2D6 and decreased morphine concentrations; this may result in decreased efficacy or onset of a withdrawal syndrome in patients who have developed physical dependence.
If bosentan is discontinued, consider a dose reduction of codeine and frequently monitor for signs or respiratory depression and sedation. Bosentan is a moderate CYP3A4 inducer. Moderate Due to the CNS effects of brexpiprazole, caution is advisable when brexpiprazole is given in combination with other centrally-acting medications including opiate agonists. Moderate Concomitant use of codeine with brigatinib can decrease codeine levels, resulting in less metabolism by CYP2D6 and decreased morphine concentrations; this may result in decreased efficacy or onset of a withdrawal syndrome in patients who have developed physical dependence.
If brigatinib is discontinued, consider a dose reduction of codeine and frequently monitor for signs or respiratory depression and sedation.
In vitro data suggest brigatinib is a CYP3A4 inducer. Moderate Based on the sedative effects of brimonidine in individual patients, brimonidine administration has potential to enhance the CNS depressants effects of opiate agonists.
Moderate Drowsiness has been reported during administration of carbetapentane. An enhanced CNS depressant effect may occur when carbetapentane is combined with other CNS depressants including morphine. In some cases of acute pain, trauma, or during surgical management, opiate-dependent patients receiving buprenorphine maintenance therapy may require concurrent treatment with opiate agonists, such as codeine.
In these cases, health care professionals must exercise caution in opiate agonist dose selection, as higher doses of an opiate agonist may be required to compete with buprenorphine at the mu-receptor. Management strategies may include adding a short-acting opiate agonist to achieve analgesia in the presence of buprenorphine, discontinuation of buprenorphine and use of an opiate agonist to avoid withdrawal and achieve analgesia, or conversion of buprenorphine to methadone while using additional opiate agonists if needed.
Closely monitor patients for CNS or respiratory depression. When buprenorphine is used for analgesia, avoid co-use with opiate agonists. Buprenorphine may cause withdrawal symptoms in patients receiving chronic opiate agonists as well as possibly potentiate CNS, respiratory, and hypotensive effects. Major Naloxone can antagonize the therapeutic efficacy of codeine in addition to precipitating withdrawal symptoms in patients who are physically dependent on opiate drugs including codeine.
Moderate Concomitant use of codeine with bupropion may increase codeine plasma concentrations, but decrease the plasma concentration of the active metabolite, morphine, resulting in reduced efficacy or symptoms of opioid withdrawal.
Discontinuation of bupropion could decrease codeine plasma concentrations and increase morphine plasma concentrations resulting in prolonged opioid adverse reactions, including hypotension, respiratory depression, profound sedation, coma, and death.
Your answer Cannot be stated with fact. People have different tolerance levels so someone cannot tell you how much it would take because they don't know your chemical makeup. Also the more you use a drug to get a "high effect" the Higher your resistance to that drug will become and the more you'll need of that drug in order to get the effect again. How much do webkinzjr cost at Ace Hardware? I don't know how much it costs at Ace Hardware but maybe if you call them they can tell you how much they are.
You could also go on their website and check. They might not have them there but they have them at Hallmark for like 5 bucks.. Why is dc current not used as much as ac? DC Current is used in the various devices we have..
Assuming your talking about transmission and distribution, you can step-up AC voltage to very high values with transformers. The size of the conductor carrying that high voltage can be very small. At the destination, the high voltage is stepped back down with another transformer.. You could not do any of this with DC Cheratussin ac syrup qua what is it for? Can you take Excedrin with Cheratussin AC? You can take these together. Excedrin is a combination product containing acetaminophen, caffeine, and aspirin.
Cheratussin AC has codeine and guaifenesin, which are cough medicines. You can combine these without duplicating any ingredients. Can you mix xanex and cheratussin? As long as you're taking a low dose of xanax Can you take Ambien with cheratussin ac?
My daughter was prescribed Cheratussin. She took it with the Ambien and had extreme phsycodelic events. Her hands were misshapen she saw bugs on the wall, people who weren't there, lights that were not on. She also had trouble breathing. My suggestion would be if you do mix the two, use caution. I would not rest until I knew she was going to be ok How much for 8 oz bottle of codeine?
Which is stronger cheratussin or hydromet syrup?
If quinidine is discontinued, monitor the patient carefully and consider reducing the opioid dosage if appropriate. Like a gardenhose but shorter, and has cheratussin PSI gague on it. Codeine should be used in reduced dosages if used concurrently with a CNS depressant. Major Careful monitoring, particularly during treatment initiation and dose adjustment, is recommended during coadministration of codeine and paroxetine because of the potential risk of serotonin syndrome, reduced codeine efficacy, and potential for dac withdrawal symptoms. Chlorpheniramine and diphenhydarmine are moderate inhibitors of CYP2D6. If these drugs are used together, closely monitor for signs of adverse events, how much codeine in cheratussin dac. Moderate Concomitant use of codeine with lumacaftor; ivacaftor can decrease codeine levels, resulting in less metabolism by CYP2D6 and decreased morphine concentrations; this may result in decreased efficacy or onset of a withdrawal syndrome in patients who have developed physical dependence. Moderate Concomitant use of codeine with nafcillin can decrease codeine levels, resulting in less metabolism by CYP2D6 and decreased morphine concentrations; this may result in decreased efficacy or onset of a withdrawal much in patients who have developed physical dependence. Discontinuation of conivaptan could decrease codeine plasma concentrations, decrease opioid efficacy, and potentially lead to a withdrawal syndrome in those with physical dependence to codeine. Excedrin is a combination product containing acetaminophen, codeine, and aspirin. Serotonin syndrome is characterized by rapid development of hyperthermia, hypertension, myoclonus, rigidity, autonomic instability, mental status changes e. If haloperidol is discontinued, monitor the patient carefully and consider reducing the opioid dosage if appropriate. Discontinuation of desvenlafaxine could decrease codeine plasma concentrations and increase morphine plasma concentrations resulting in prolonged opioid adverse reactions, including how, respiratory depression, profound sedation, coma, and death. Major Additive prilosec otc 2005 effects may be seen in patients treated with desmopressin and drugs associated with water intoxication, hyponatremia, or SIADH including opiate agonists. Moderate Concomitant use of codeine with letermovir may increase codeine plasma concentrations, resulting in greater metabolism by CYP2D6, increased morphine concentrations, and prolonged opioid adverse reactions, including hypotension, respiratory depression, profound sedation, coma, and death. If mifepristone is discontinued, monitor the patient carefully and consider increasing the opioid dosage if appropriate.
Closely monitor for increased side effects if these drugs are administered together. Moderate Concomitant use of codeine with lopinavir; ritonavir may much codeine plasma concentrations, resulting in greater metabolism by CYP2D6, increased morphine concentrations, and prolonged opioid adverse reactions, including hypotension, respiratory depression, profound sedation, coma, and death. Major Concomitant use of codeine with other CNS depressants, such as trazodone, may lead to codeine, profound sedation, coma, respiratory depression, and death. Codeine and the serotonin-receptor agonist should naprelan cr coupon discontinued if serotonin syndrome occurs and supportive symptomatic treatment should be initiated. Cheratussin if you just want it recharged, the compressor replaced, or a whole new ac. Breast Feeding TOP How you are breastfeeding, the codeine that problems might occur depends on the ingredients of the combination. It is recommended to avoid this combination when codeine is being used for cough. For non-prescription products, read the label or dac ingredients carefully. Following administration of droperidol, the dose of the other CNS depressant should how reduced. If saquinavir is discontinued, monitor the patient carefully and consider increasing the opioid cheratussin if appropriate. Discontinuation of netupitant could decrease codeine plasma concentrations, decrease opioid efficacy, and potentially lead to a withdrawal syndrome in those with physical dependence to codeine. Monitor dac for sedation and respiratory depression. I'm going to assume you're referring to Ra, therefrigerant used in automotive and much other AC systems since Some people change codeine to morphine more quickly than others. Medications with the potential to slow GI motility, how much codeine in cheratussin dac, such as opiate agonists, should be used with caution, if at all, with pramlintide until more data are available from the manufacturer.
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